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Clinical characteristics of AITL patients. A total of 173 newly diagnosed patients with AITL were included and analyzed in this study. The baseline clinical characteristics of the patients are ...

发病机制：aitl的tfh细胞起源理论得到充分印证，突变细胞通过分泌cxcl13促进滤泡树突细胞（fdcs）增殖，形成特征性高内皮微静脉（hevs）。本例中ebv感染与cxcl13 + 细胞共现，符合aitl的"双打击"发病模型。 鉴别诊断：血管性水肿的病因学分析成为亮点。

rhoa g17v突变被视为aitl的分子标签，与tet2突变显著相关（p. 0.05）。. 染色体异常以5号染色体三体最为特征性，与idh2 r172突变存在显著关联（p=0.01）。

Poster Title: ARV-393, a PROteolysis TArgeting Chimera (PROTAC) BCL6 Degrader, is Efficacious in Preclinical Models of Diffuse Large B-Cell Lymphoma, Nodal T-Follicular Helper Cell Lymphoma, and ...

– ARV-393 demonstrated significant single-agent activity in models of nodal T-follicular helper cell lymphoma, angioimmunoblastic-type (also known as AITL) and transformed follicular lymphoma ...

“We are encouraged by the marked single-agent activity of ARV-393 in PDX models of AITL and transformed follicular lymphoma and by the enhanced antitumor activity of ARV-393 in combination with five ...

ARV-393 demonstrated significant single-agent activity in a patient derived xenograft (PDX) model of nodal T-follicular helper cell lymphoma, angioimmunoblastic-type (nTFHL-AI also known as AITL ...